Oxyresveratrol CAS 29700-22-9
Chemical Name: Oxyresveratrol
Synonyms: trans-Oxyresveratrol
CAS No.: 29700-22-9
Molecular Fomula: C14H12O4
Molecular weight: 244.24
Appearance: White to light yellow powder
Assay: 98%
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Description
Oxyresveratrol Quick Details
Chemical Name: Oxyresveratrol
CAS No.: 29700-22-9
Molecular Fomula: C14H12O4
Chemical Structure:
Molecular weight: 244.24
Appearance: White to light yellow powder
Assay: 98%
Typical Properties
ITEMS
SPECIFICATION
Density
1.468??0.06 g/cm3(Predicted)
Boiling point
523.8??30.0 ??C(Predicted)
Melt point
201-202.5 ??C
?
Oxyresveratrol?application:
Antioxidant, with anti-inflammatory, antithrombotic, anti-cancer, anti-cancer, anti-hyperlipidemia, antibacterial and many other activities
Packaging and Shipping
25kg/drum
Oxyresveratrol?Storage
This product shall be airtight, out of the sun, moisture-proof and away from heat. The storage period is one year.
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- Ammonium sulfamate
- Benzothiazole
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- SODIUM ETHYL 2-SULFOLAURATE
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Chemical Name: Oxyresveratrol
Synonyms: trans-Oxyresveratrol
CAS No.: 29700-22-9
Molecular Fomula: C14H12O4
Molecular weight: 244.24
Appearance: White to light yellow powder
Assay: 98%
Huperzine A is a natural plant extract. Compared with similar AD treatment drugs approved by the US FDA, Huperzine A has a unique chemical structure and has extremely high selectivity to inhibit acetylcholinesterase in the brain and enhance cholinergic in the brain. function of neurons. In addition, Huperzine A has obvious effects on improving memory and improving cognitive ability.
Huperzine A has a small relative molecular weight, high lipid solubility, and is easy to pass through the blood-brain barrier. After entering the central nervous system, it is mostly distributed in the frontal lobe, temporal lobe, hippocampus and other parts of the brain. Its pharmacological effects have multiple targets. In addition to inhibiting the activity of acetylcholinesterase, it can also antagonize oxidative stress and apoptosis induced by ??-amyloid peptide (A??), hydrogen peroxide and other neurotoxins, by activating the protein kinase C (PKC) signal transduction pathway. Activation of ??-secretase promotes the decomposition of ??-amyloid precursor protein (APP) in a non-amyloidogenic manner to produce sAPP??, reducing A??-mediated toxicity, while sAPP?? can effectively promote cell proliferation, axonal growth, and protect nerves. cell.
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